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Accordingly, the proinflammatory potential of BPA has been pointed out as a mechanism of obesogenic disruption, activating proinflammatory signalling in fat cells by upregulating the expression and release of specific cytokines, such as CCL20, IL-18, IL-6, IL-1, and TNF- [174, 175]

Another significant function attributed to ICP6 involves its modulation of programmed cell death (PCD) processes in infected cells through its receptor-interacting protein-homotypic interaction motif (RHIM) (54)