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GLP-1 is secreted by L-cells in the intestine in response to nutrient sensing, particularly glucose, amino acids and certain fats

The compound crosses the blood-brain barrier efficiently after oral administration and demonstrates sustained cognitive benefits even after treatment cessation, suggesting true structural enhancement rather than temporary stimulation

Such findings suggest a possible role in maintaining circulation during tissue damage

Importantly, although patient perceptions of deformity may not predict objective measures of deformity, and patient distress is poorly correlated with objective measures of deformity (Hellstrom 2013), patient perceptions of deformity correlate with distress