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Focus on the primary prevention of intrauterine adhesions: current concept and vision [J]

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This paper reviews the Met metabolism in normal conditions and its dysregulation under abnormal conditions, the possible causes of hypermethioninemia, and its connection to BA pathogenesis: Abnormal hepatic level of Met could lead to a perturbation of redox homeostasis and mitochondrial functions of hepatocytes, enhancement of viral infectivity, and dysregulation of innate and adaptative immune cells in response to infection/damage of the liver contributing to the initiation/progression of BA

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doi: 10.1097/CCM.0b013e31819305be 37 LemichezE.AktoriesK

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