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Thus, for ferroptosis to proceed, PUFAs were required to intersect into membrane to serve as the targets for peroxidation and iron-catalyzed free radical production

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It activates the HGF/Met signaling pathwaypromoting synaptogenesis and dendritic spine formationat picomolar concentrations that exceed brain-derived neurotrophic factor and other nootropic compounds by several orders of magnitude

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New blood vessel formation brings oxygen, nutrients, and immune cells to injured tissues, addressing the fundamental bottleneck in tendon and ligament healing where poor vascularization slows repair