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Conversely, stressed or dying chondrocytes release DAMPs, apoptotic bodies, cartilage fragments, and pro-inflammatory mediators that further activate synovial macrophages and fibroblasts, maintaining a chronic synovitic state and perpetuating pain and swelling (Fan et al., 2024

TAMs have an inflammatory phenotype in the early stages of cancer formation and an immunosuppressive phenotype in the later stages of cancer progression [57]

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