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Medications such as valproic acid, cyclosporine, pivampicillin, and some anti-cancer drugs (e.g., etoposide, vinblastine) can also contribute to carnitine deficiency.[6] Valproic acid therapy may deplete carnitine stores by various mechanisms such as increased urinary excretion as valproylcarnitine, decreased renal tubular reabsorption, and decreased endogenous production
Polyphenols, terpenoids, carotenoids, marine and herbal products, as well as vitamins and animal-derived molecules, can counteract oxidative damage, modulate inflammatory cascades, and restore nerve conduction and pain
demonstrated that ethanol or acetaldehyde-induced ferroptosis was closely associated with the autophagic degradation of nuclear receptor coactivator 4 -dependent ferritin
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