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Regenerative and protective actions of the GHK-Cu peptide in tissue repair
Neuroprotection : The peptide counteracts neurotoxic damage by modulating dopaminergic and serotonergic pathways in the brain and peripheral nervous system
The mechanism involves upregulation of growth hormone receptors in tendon fibroblasts and increased collagen synthesis at the repair site
The peptide may no longer bind to its target receptor
In fact, several related observations suggest that endogenous melatonin acts to counter undesirable abrupt changes in phase
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