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Limiting inflammation-the negative regulation of NF-kappaB and the NLRP3 inflammasome

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Additionally, angiotensin II contributes to endothelial dysfunction by inducing expression of vascular endothelial growth factor and increasing reactive oxygen species, which reduce the bioavailability of vasodilators (such as nitrous oxide) responsible for maintaining normal glomerular vascular tone [14]

The findings do not undermine the efficacy of the drugs, experts say, but they need to be carefully considered by those paying for them and by patients considering their treatment options