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Data adapted from Knig's research group at the University of Vienna

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Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)

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It mainly looked at the combination of semaglutide and cagrilintide, but also included groups on cagrilintide alone and on placebo

doi: 10.1016/j.tim.2020.02.004 13 FrancoATJohnstonEKrishnaUYamaokaYIsraelDANagyTAet al