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Assays of endothelial cell migration showed that BPC-157 enhanced the chemotactic response of endothelial cells

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In normal physiologic state, the brain parenchyma is only subject to nascent immune surveillance, however during inflammation, the elevated pro-inflammatory markers induce the increased expression of adhesion molecules like intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule-1 (VCAM-1) on the endothelial cells of the BBB (Daneman and Prat, 2015

The acylated side chain and amino acid substitutions extend half-life in vivo and also improve storage stability