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The mutant huntingtin protein can disrupt mitochondrial function and increase ROS production

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therefore, tau and TSPO PET were not completed as part of this study, and cerebrospinal fluid data were also not collected

As direct peroxisome proliferator-activated receptor a (PPAR)- regulated genes, CPT1A, CPT2 and LCAD were up-regulated after treatment with PPAR agonist, fenofibrate (Feno), indicating the improvement of -FAO

I want to be clear about this because I hear the misconception often: Foundayo is not a lesser option or a starting rung on a ladder