Researchers observed that BPC-157 administration was associated with maintained gastric mucosal integrity and altered expression of protective mucosal factors compared to vehicle controls [1]
Besides acetylcholine (ACh) receptors, microglia also expressed choline transporter CTL1, primarily located in the plasma membrane, facilitating extracellular choline transport, and CTL2 mainly in the mitochondria, suggesting a role in betaine production (99)
There were no SAEs requiring withdrawal reported during the whole treatment period in any group
Boughton CK, et al
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