It drives the transition from proliferation to differentiation and increases the number and length of multinucleated myotubes in a dosedependent manner.The mechanism is mainly related to NNMT inhibition, restored NAD levels, and activation of the SIRT1PGC1 pathway
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doi: 10.1210/er.2006-0021
People can also have 'normal' levels of B12 in their blood, but be symptomatic - this is known as a functional B12 deficiency
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98% purity verified by HPLC