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However, tumor cells rely on cell cycle checkpoints to gain time to repair DNA damage caused by radiation exposure, resulting in the resistance to radiotherapy

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Ultimately, this FOXO4-DRI-mediated mechanism selectively eliminates senescent endothelial cells, which in turn attenuates vascular inflammation and improves endothelial-dependent vasodilation, conferring a significant protective effect on vascular function

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The exact mechanism by which GSH helps in maintaining normoglycemia in diabetic patients requires further investigation