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Our findings, combined with existing evidence, suggest that semaglutide and perhaps other GLP1-RAs should be reconceptualized as disease-modifying treatments rather than solely medications for glycaemic control or weight loss, concluded the authors

doi: 10.3164/jcbn.12-112 38 AyalaAMuozMFArgellesS

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Applying the same ionizable scaffold to semaglutide preserved peptide integrity and normalized fasting glucose and homeostasis model assessment of insulin resistance (HOMA-IR) in Western-diet mice within 48 h, whereas 1,2-distearoyl-sn-glycero-3-phosphocholine (DSPC)-based liposomes did not reach significance [118]