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Han X, Chen L, Guo L, et al

When modified by NAPQI, Keap1 releases Nrf2, enabling its translocation into the nucleus, where it binds to antioxidant response elements in DNA and promotes transcription of genes encoding various antioxidant enzymes

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While most research pertaining to glutamate disruption and cognitive dysfunction in schizophrenia has focused on the NMDA receptor [9,10,11], recent attention has turned to manipulations that target glutamate synthesis, release or reuptake

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