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Superficial adhesion of planktonic cells primarily contact substrates of the host organism during infection with various adhesins, such as fibrinogen, fibronectin and collagens such as fibronectin binding proteins (FnBPs) and fibrinogen-binding clumping factors (Clfs) (Palmqvist et al., 2005)

The relation between calcium buffering capacity and calcium dynamics has been demonstrated in CA1 str

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