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The medication operates by diminishing appetite and reducing food intake

Marie-Christine Beauvieux, CNRS Universit Bordeaux, France Copyright 2015 Spreckley and Murphy

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By bridging rigorous mechanistic insights with real-world therapeutic applications, we aim to underscore how a deep understanding of GLP-1 biologyspanning metabolic, cardiovascular, inflammatory, and neurohormonal pathwayscan inform more precise, effective, and individualized treatment strategies

It reduced pro-inflammatory cytokines (TNF-, IL-6, IL-1), chemokines, adhesion molecules and oxidative stress, while upregulating the expression of neurotrophic factors (neuritin and NGF) [11]

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