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left) and its interaction with the extracellular domain of GLP-1R (PDB: 3IOL

Humana Press, 437-445, 1987

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(172) demonstrated that intravenous infusion of porcine GIP abolished plasma hypertriglyceridemia in rats during the fat load, while rats pretreated with GIP antiserum exhibited a significantly greater triglyceride increment late in the fat load time course

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