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Despite the pathophysiological and clinical differences between AD and PD, the basis for the use of GLP-1RAs in the treatment of these diseases is precisely the general mechanisms of their pathogenesis and risk factors, including T2DM and insulin resistance, abnormal accumulation of toxic proteins, increased inflammation, decreased synaptic function, loss of neurons, increased activation of astrocytes, leading to cognitive dysfunction [43]
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The presence of high levels of circulating trimethylamine N-oxide exacerbates central and peripheral inflammation and inflammatory hyperalgesia in rats following carrageenan injection
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