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Notably, metabolomics studies have demonstrated that LH stimulation rapidly alters the metabolic profile of luteal cells, involving remodeling of multiple metabolic pathways, including glycolysis, the TCA cycle, the pentose phosphate pathway, and lipid biosynthesis [73]

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Unlike older compounds in its class, it specifically targets the growth hormone secretagogue receptor without significantly elevating cortisol, prolactin, or ACTH levels during assays

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