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The answer may lie, in part, in the body's ability to manage severe oxidative stress, repair mitochondrial damage, and efficiently clear cellular debris

The authors suggest an implication of the phosphorylated CREB, mediated by PI-3K and MAPK signaling pathways, that is known to decrease neurotoxicity and oxidative stress-induced neuronal death (Riccio et al., 1999

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& Al-Mansour, M

Cortical parvalbumin interneurons and cognitive dysfunction in schizophrenia