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Evidence from experimental models of emphysema and from individuals with genetic deficiency of alpha-1 antitrypsin provides strong evidence that an imbalance between the enzymes and inhibitors is important in tissue damage and the pathogenesis of COPD

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Polydatin promotes Nrf2-ARE anti-oxidative pathway through activating Sirt1 to resist AGEs-induced upregulation of fibronetin and transforming growth factor-1 in rat glomerular messangial cells

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