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While the SNSs direct effect on reward sensitivity requires further investigation, the metabolic activation from glucagon may complement the direct reward-suppressing effects of GLP-1 and GIP pathways in SUD treatment

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This outperformance of an established clinical therapy suggests significant potential for diabetic wound applications

#cancerawareness To view or add a comment, sign in Gladstones Alex Marson, along with collaborators at Arc Institute and UCSF, developed a way to create more effective T cell therapies that can potentially treat cancer, autoimmune diseases, and other conditions