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However, during hyperglycemia, cotadutide, liraglutide, and vehicle control groups responded with normal increases of insulin secretion, while g1437-treated mice were unable to mount an insulin secretory response

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Legault Z, Bagnall N, Kimmerly DS

Key data indicate that knocking out GIPR in GABAergic (VGAT+) neurons ablated the ability of GIPR mono-agonists to drive weight loss on their own and for dual GLP-1R/GIPR agonists to produce additive weight loss beyond GLP-1R agonists alone [31]

- 24 hours for reconstituted with sterile water

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