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Its mechanism of action is reflected in two aspects: first, SESN2 regulates the AMPK/mTORC1 pathway

Importantly, we further identified HO-1 mitochondrial translocation as a key mechanism for oxidative stress-induced mitochondrial iron overload and ROS accumulation

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vii) no serious concomitant medical conditions mimicking AAV such as malignancies, severe infectious diseases requiring hospitalization, and autoantibody-medicated autoimmune diseases, or inducing ANCA false positive at diagnosis