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In a mouse model of MS, UA can penetrate the compromised blood-brain barrier (BBB), block ONOO -mediated tyrosine nitration, and contribute to the containment of the central nerve system (CNS) pathology [44]

MSU crystals, infiltrating macrophages, and macrophage-derived cytokines create a specialized microenvironment that induces the conversion of resting fibroblasts into activated myofibroblasts

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