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doi: 10.1146/annurev-nutr-071714-034250 197 SebaiHHovspianSRistorcelliEAouaniELombardoDFayetG
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Common mutations in p53, including missense, nonsense, and frameshift mutations, can increase the proteins half-life, leading to the accumulation of dysfunctional mutant p53 that impairs normal tumour-suppressive functions (37, 38)
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