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Gene alterations modulating glutamine dependency in cancer The dependency of glutamine metabolism in cancer is not simply a passive adaptation to proliferative state, but rather a tightly regulated response to a multitude of intracellular and extracellular factors, including potential genetic alterations

The unveiling of SLC1A5, SLC7A5, SLC6A14, and the SLC38 family transporters, along with their distinctive functional characteristics, enriches our comprehension of the intricate nature of glutamine transport in cancer

A possible hypothesis is that aggregation is probably a consequence of oxidative modications of -synuclein [69]

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