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The peptide protects gastric mucosa from damage induced by NSAIDs (non-steroidal anti-inflammatory drugs), alcohol, and stress, counteracting the pathological effects of these ulcerogenic agents

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Later on, additional roles for the antioxidant function of GSH have emerged that are strictly related to signal transduction: (i) the interaction of the tripeptide with NO or with RNS (see next section)

et al., oral administration of Lactiplantibacillus plantarum Y15 to STZ + HFD-induced T2D mice improved fasting glucose, HbA1c, and HOMA-IR levels, reduced pro-inflammatory cytokines (IL-6, TNF-, among others), decreased the abundance of LPS-producing bacteria, and promoted SCFA-producing populations