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A role for VIP in the development of asthma has also been demonstrated in studies involving mice lacking the VIP gene that exhibited spontaneous asthma features, such as peribronchial airway inflammation and the production of pro-inflammatory cytokines and AHR, which was partially reduced by administration of exogenous VIP [95]

Crizotinib induced apoptosis and greatly decreased the tumor burden in a patient derived xenograft in vivo model