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Nitric oxide (NO), a specific oxygen free radical synthesized by nitric oxide synthase (NOS), significantly increases in injured rat and human rotator cuff tendons compared to healthy tendons [37]

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While it has been in research for over 20 years, it has recently gained a second life thanks to the massive public interest in peptide therapy and the rise of GLP-1 medications.

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166 A recent study reported that S-nitrosylation altered AAT to a proinflammatory protein, which facilitated the inflammatory state of macrophages by upregulating proinflammatory molecules and the activation of MAPK signalling pathways