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Comprehensive Psychiatry

626 The absence of ALOX5 expression eliminates HTTQ94-induced ferroptosis under oxidative stress conditions, suggesting that ALOX5 is essential for ferroptosis mediated by mutant huntingtin and could be a promising new target for HD treatment

doi: 10.1016/j.ymgme.2015.10.003 88 StocklerSSchutzPWSalomonsGS