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At the level of the microbiotaimmuneneural interface, DA and NE not only act as neuromodulators but also influence bacterial growth and virulence, creating bidirectional feedback between catecholaminergic tone and microbial composition [209,210]

88.9%), and therapeutic inertia was observed in only 45 individuals (11.1%) (Table 6)

Disrupted Cacna1c gene expression perturbs spontaneous Ca(2+) activity causing abnormal brain development and increased anxiety

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