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These check-ins allow Dr
The results of LDSC analysis show weak evidence of a genetic correlation between SLE and ursodeoxycholate ( rg = 0.0395, se = 0.2454, p = 0.8722), taurodeoxycholate ( rg = 0.1601, se = 0.1715, p = 0.3503), N1-methyladenosine ( rg = 0.0427, se = 0.362, p = 0.7538), 1,5-anhydroglucitol (1,5-AG) ( rg = 0.0395, se = 0.0840, p = 0.6383), 1-arachidonoylglycerophosphocholine ( rg = 0.0320, se = 0.1026, p = 0.7554), isobutyrylcarnitine ( rg = 0.1165, se = 0.1219, p = 0.3392), palmitoleate (16:1n7) ( rg = 0.2461, se = 0.1551, p = 0.1124), and 1-oleoylglycerophosphoethanolamine ( rg = 0.1058, se = 0.2077, p = 0.6105), suggesting that the shared genetic component did not confound the MR estimates (Supplementary Table S1)

In vitro studies by Wang et al., using HUVEC cells, suggest that GHK-Cu may stimulate the expression of vascular endothelial growth factor (VEGF) and fibroblast growth factor-2 (FGF-2), both of which are well-characterized mediators of endothelial cell proliferation and new vessel formation
This improves blood flow to damaged tissue, allowing nutrients, oxygen, and immune cells to reach the injury site more efficiently, speeding up recovery (Gwyer et al., Frontiers in Physiology )
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