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Another mitochondrial toxin and complex I inhibitor, 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine, triggers parkinsonism in humans [93]

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This indicated that the amount of GSH increased significantly due to active nitrogen fixation in wild-type nodules and enhanced defense responses in the nodules of ineffective mutants ( FIGURE 4 TABLE 2 The distribution of immunogold-labeled glutathione in regions of infected cells in pea wild-type and mutant nodules

J Am Heart Assoc 11:e027228

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