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The primary pathogenic mechanism contains genetic alternations, hormonal homeostasis changes, and immune interference, which are demonstrated as follows

for example, salinomycin can drive ferroptosis-based cell death in breast CSCs (Zhao et al., 2019), and ironmycin, a derivative of salinomycin, can specifically trigger iron accumulation in lysosomes, activating cell death pathways consistent with ferroptosis (Mai et al., 2017)