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Notably, the developmental defects in the hdip1/2/3 mutant were significantly complemented by the HDIP1-Flag transgene (Appendix Fig

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Our observation of a widespread induction of pathways required for viral replication in uninfected cells, in combination with the previous detection of extracellular vesicles containing viral proteins in SARS-CoV-2 infected individuals 62,63 indicate that SARS-CoV-2 infected cells might modulate the permissibility of their microenvironment for viral replication in a similar manner