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Treatment significantly attenuated both clinical and histological signs of arthritis

Furthermore, increased astrocytes levels of miR-155 were shown to target SOCS1 , a negative regulator of the inflammatory gene response, in A-treated astrocytes, causing prolonged expression of inflammatory cytokines (Guedes et al., 2014)

For all-cause mortality, the incidence was noted to be 16.1% in the highest quartile group compared to 6.3% in the lowest quartile group (HR = 2.21) [27]