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Until such data exist, the current results should be regarded as hypothesis-generating and a useful warning against indiscriminate use of glutathione in chronic liver disease

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In PD models, Stat1 ablation alleviated -synuclein fibril-induced suppression of microglial ULK1 transcription and autophagy, which is consistent with the mechanism whereby -synuclein disrupts microglial autophagy through STAT1-dependent repression of ULK1 expression