Most of the dosing protocols circulating in the peptide community are educated extrapolations based on animal data and anecdotal human experimentation
For example: Chronic mild stress inhibits mitochondrial function at respiratory chain complexes I, III, and IV.[ref] Lets dig into chronic stress further: Chronic stress: a cause of mitochondrial dysfunction
What is missing from many patients is better education about how these medications work, strategies to manage side effects on a personal level, and solutions to make them less expensive
Preclinical and in vitro work suggests that GHK-Cu can modulate collagen and glycosaminoglycan turnover, influence fibroblast and immune-cell behavior, and interact with pathways related to oxidative stress and tissue injury signaling
These experiences, while not universal, suggest that the refund process could be more patient-friendly
P., Folker, J