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However, while these experiments did not distinguish which KCC may be involved in the stimulatory effects of high doses of the diuretic, inhibition of -cell KCC2 with highly selective drugs (Kursan et al., 2017) or its transient siRNA-mediated downregulation in islets (Pae and Harper, 2021) resulted in increased insulin secretion in response to glucose

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Diverse rescue potencies of p53 mutations to ATO are predetermined by intrinsic mutational properties

We examined metabolomic profile changes that may explain increased fatigue severity in response to receiving ADT (Figure 7)

Rudman D, Sewell CW, Ansley JD: Deficiency of carnitine in cachectic cirrhotic patients

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