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On the gastrointestinal front, literature has described mucosal barrier protection via modulation of endogenous prostaglandin (PGE2) and heat shock proteins (HSP70/90)

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showed the benefits and risks of Gln supplementation, while clinical trials in burns and other diseases have shown conflicting results

However, the precise molecular mechanisms involved in this phenomenon and the putative clinical implications of this T-cell regulation remain to be determined, as some discrepancies exist between in vivo and in vitro findings

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Type 2 diabetes: sensitization, not just weight Diabetes trials measure a different baseline and endpoint than obesity trials