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Nat Rev Neurol 15:459472
[DOI] [PubMed] [Google Scholar] Gonzlez SRangel-Barajas CPeper MLorenzo RMoreno ECiruela FBorycz JOrtiz JLlus CFranco R, et al
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*Correspondence: Prachi Pandey, International Center for Genetic Engineering and Biotechnology, Aruna Asaf Ali Marg, New Delhi, Delhi 110067, India [email protected] This article was submitted to Environmental Toxicology, a section of the journal Frontiers in Environmental Science Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
However, many of these effects seem to be mediated via -adrenergic receptors (Parrado et al., 2012, 2017), and with the contrasting findings on the role of D1- and D2-like activation, the specific processes by which dopamine could influence keratinocyte activity to drive psoriasis remain unclear
