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When glutathione levels are inadequate, the immune system can become dysregulated, either becoming overactive (increasing inflammation and risk of autoimmune disorders) or underactive (reducing the body's ability to fight infections)
Excess copper is reduced by FDX1 to generate Cu, which drives cell death via two primary mechanisms: (1) Cu-mediated aggregation of lipoylated TCA cycle proteins (e.g., DLAT) and loss of Fe-S clusters, and (2) Cu-induced disruption of the Npl4/p97 protein quality-control system
10.1016/j.bbrc.2009.07.003 Biochem
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