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Glutathione depletion renders hepatocytes vulnerable to TNF-alpha-induced apoptosis, contributing to alcoholic hepatitis [6][35]

Knockout of the Mouse Glutamate Cysteine Ligase Catalytic Subunit (Gclc) Gene: Embryonic Lethal When Homozygous, and Proposed Model for Moderate Glutathione Deficiency When Heterozygous

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APAP administration promotes apoptotic-response and regenerative in the adult fat body In vertebrate systems, APAP administration is associated with massive necrosis, possible apoptosis, and a prompt regenerative response 33,34