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Yet another mechanism contributing to the specific vulnerability of the gastrointestinal system to TD may involve the gut-brain axis, which depends on vagal tone and acetylcholine neurotransmission, for which thiamine co-release is known (165167)

UA plays a crucial role in improving mitochondrial function by stimulating mitophagy and inhibiting the age-related accumulation of dysfunctional mitochondria, thereby maintaining mitochondrial biogenesis and respiration within cells (1)

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