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Conversely, deletion of FTMT further promoted tBHP-induced cell death in cardiomyocytes (Fig

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Critical evaluation Strengths - Addresses unmet need for disease modifying diabetes therapies, also applicable to related metabolic diseases - Combines regenerative potential with metabolic control - Supported by robust preclinical models and biomarker endpoints Limitations - Translational challenges in moving from preclinical models to human cells regeneration - Long term safety must be carefully evaluated, given its role in gastric physiology - Regulatory guidelines for such therapies are still evolving

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