These effects have been particularly relevant in neurodegenerative conditions and metabolic disorders, where mitochondrial dysfunction plays a central role
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Induction of ferroptosis by erastin, C dot, or FAC and BSO leads to its dissemination to neighboring cells through an iron and lipid peroxide-dependent mechanism, whereas the GPX4 inhibitor ML162 fails to transmit the ferroptosis signal to adjacent cells
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After blocking the ferroptosis pathway within tumor cells, their sensitivity to immunotherapy is lost
