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Patients have variable numbers of dysplastic nevi, ranging from a few to hundreds of lesions

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The expression changes in miRNAs alter the expression of their target genes, potentially decreasing the activity of oncogenes as well as increasing that of tumor suppressor and DNA repair genes [32]

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In this process, DHODH functions concurrently with the mitochondrial GPX4, separate from the cytoplasmic GPX4 and FSP1 (55)